Ever wonder why SARS-CoV-2 was so successful in infecting different people, even those who were otherwise healthy and had a robust immune system? Part of the reason could be the ability of the virus to dampen the anti-viral innate immune response by impairing the mechanism of interferon production in host cells. In collaboration with Tommy Alain's team at University of Ottawa, we wrote a mini-review that distills the latest findings on all the ingenious ways through which SARS-CoV-2 block interferon production in host cells. Find the paper here and enjoy reading!
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See AllWe are thrilled to see the new paper by our collaborator Costa's group, featuring our very own postdoc Parisa as a contributor! This paper reports an exciting mechanism of regulation of sprouting angi
We are thrilled to announce Susanta’s latest achievement in publishing his latest groundbreaking findings in a preprint on bioRxiv! Susanta's work has unravelled a crucial role for the Ribosome Qualit
It is sometimes difficult to tease apart the molecular functions and biological roles of paralog proteins. In this preprint we do just that for GIGYF1, a protein that has for years been overshadowed b
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