Ever wonder why SARS-CoV-2 was so successful in infecting different people, even those who were otherwise healthy and had a robust immune system? Part of the reason could be the ability of the virus to dampen the anti-viral innate immune response by impairing the mechanism of interferon production in host cells. In collaboration with Tommy Alain's team at University of Ottawa, we wrote a mini-review that distills the latest findings on all the ingenious ways through which SARS-CoV-2 block interferon production in host cells. Find the paper here and enjoy reading!
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See AllCongratulations to our collaborators at Sossin lab (Montreal Neurological Institute) and Gkogkas lab (Foundation for Research and Technology-Hellas) for publication of their very enticing new paper on
We are starting the new year with the excellent news of Parisa's new paper in bioRXiv . We had shown in the past that the cap-binding protein 4EHP is a critical component of the mechanism that mediate
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